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Pablo Lazcano
Pablo Lazcano
Research Assistant, Universidad de los Andes
Verified email at uandes.cl
Title
Cited by
Cited by
Year
TNF-α increases production of reactive oxygen species through Cdk5 activation in nociceptive neurons
R Sandoval, P Lazcano, F Ferrari, N Pinto-Pardo, C González-Billault, ...
Frontiers in Physiology 9, 328235, 2018
592018
Targeted overexpression of tumor necrosis factor-α increases cyclin-dependent kinase 5 activity and TRPV1-dependent Ca2+ influx in trigeminal neurons
P Rozas, P Lazcano, R Piña, A Cho, A Terse, M Pertusa, R Madrid, ...
Pain 157 (6), 1346-1362, 2016
472016
Cardiolipin-deficient cells depend on anaplerotic pathways to ameliorate defective TCA cycle function
V Raja, M Salsaa, AS Joshi, Y Li, CWT van Roermund, N Saadat, ...
Biochimica et Biophysica Acta (BBA)-Molecular and Cell Biology of Lipids …, 2019
172019
Cyclin-dependent kinase 5 modulates the P2X2a receptor channel gating through phosphorylation of C-terminal threonine 372
C Coddou, R Sandoval, P Castro, P Lazcano, MJ Hevia, M Rokic, B Hall, ...
Pain 158 (11), 2155-2168, 2017
152017
Inositol depletion regulates phospholipid metabolism and activates stress signaling in HEK293T cells
M Suliman, KC Case, MW Schmidtke, P Lazcano, CJ Onu, ML Greenberg
Biochimica et Biophysica Acta (BBA)-Molecular and Cell Biology of Lipids …, 2022
132022
Phosphatidic acid inhibits inositol synthesis by inducing nuclear translocation of kinase IP6K1 and repression of myo-inositol-3-P synthase
P Lazcano, MW Schmidtke, CJ Onu, ML Greenberg
Journal of Biological Chemistry 298 (9), 2022
112022
NAD supplementation improves mitochondrial performance of cardiolipin mutants
J Ji, D Damschroder, D Bessert, P Lazcano, R Wessells, CA Reynolds, ...
Biochimica et Biophysica Acta (BBA)-Molecular and Cell Biology of Lipids …, 2022
102022
Valproate activates the Snf1 kinase in Saccharomyces cerevisiae by decreasing the cytosolic pH
M Salsaa, K Aziz, P Lazcano, MW Schmidtke, M Tarsio, M Hüttemann, ...
Journal of Biological Chemistry 297 (4), 2021
62021
Cardiolipin-deficient cells depend on anaplerotic pathways to ameliorate defective TCA cycle function (vol 1864, pg 654, 2019)
V Raja, M Salsaa, AS Joshi, Y Li, CWT van Roermund, N Saadat, ...
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS 1864 (8 …, 2019
12019
Abstract 2303 Cardiolipin at the epicenter of energy metabolism–implications for Barth syndrome
M Greenberg, Z Liang, T Ralph-Epps, M Schmidtke, P Lazcano, ...
Journal of Biological Chemistry 300 (3), 2024
2024
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells
Z Liang, T Ralph-Epps, MW Schmidtke, P Lazcano, SW Denis, ...
bioRxiv, 2024.02. 03.578755, 2024
2024
Anomalous peroxidase activity of cytochrome c is the primary pathogenic target in Barth syndrome
VE Kagan, YY Tyurina, K Mikulska-Ruminska, D Damschroder, ...
Nature Metabolism 5 (12), 2184-2205, 2023
2023
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